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Biological sign pinpoints 'fast agers' at high risk of kidney disease

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A new study led by Federation University researchers has identified a specific biological signature within the human kidney that could hold the key to predicting and managing age-related organ failure. The research shows that the shortening of telomeres within kidney cells, coupled with specific chemical changes to DNA, may contribute to the development of nephrosclerosis—the hardening and scarring of kidneys often associated with aging and a major contributor to chronic kidney disease (CKD).


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